Ventricular Septal Defect
Introduction
Any of the following should be reported to your child's health care provider:
-
In the first few months of life, poor weight gain or a slowdown in weight gain
- Unusual behavior
- Any additional symptoms listed in the preceding section
An immediate visit to the nearest hospital emergency department is warranted if you notice any of the following in your infant:
- Shortness of breath, breathing difficulty of any type, or worsening of an existing breathing problem
- Bluish hue of the lips, skin, or nail bed
- Unusual or unexplained sweating
Follow-up
Regular office visits and echocardiograms are required to continually reassess the
ventricular septal defect.
The child's weight and length/height will be checked often. Feeding and activity levels should be assessed routinely.
Routine antibiotic use is warranted for dental surgery and any invasive procedure.
Prevention
A woman can do nothing during
pregnancy
to prevent her baby from developing a ventricular septal defect.
Outlook
During the
growth of a child, the defect may become smaller and close on its own.
- Twenty to 25% of all ventricular septal defects close by age 3 years without medical intervention.
- Children who show no symptoms and are being monitored by a primary care provider do not have to restrict their activities. Children with mild-to-moderate shunting of blood may have to reduce their levels of activity.
- Once a defect is repaired, there are no restrictions on activity.
Several other conditions may result from ventricular septal defects.
-
Aortic regurgitation: Blood flowing backward from the aorta into the left ventricle.
- Endocarditis: An infection of the heart valves due to abnormal blood flow. Because endocarditis is always possible, medical professionals recommend that children with ventricular septal defects routinely receive antibiotics before undergoing dental procedures or surgery.
-
Pulmonary
hypertension: An increase in pressure in the right side of the heart and in the arteries of the lungs. This is caused by the shunting of blood from the left to the right ventricle, which increases the pressure in the right ventricle.