IT REDUCES HEPATIC VERY LOW-DENSITY LIPOPROTEIN TRIGLYCERIDES (VLDL-TG) SYNTHESIS AND/OR SECRETION AND ENHANCES TG CLEARANCE FROM CIRCULATING VLDL PARTICLES. POTENTIAL MECHANISMS OF ACTION INCLUDE INCREASED ?-OXIDATION; INHIBITION OF ACYL-COA:1,2-DIACYLGLYCEROL ACYLTRANSFERASE (DGAT); DECREASED LIPOGENESIS IN THE LIVER; AND INCREASED PLASMA LIPOPROTEIN LIPASE ACTIVITY.